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DGL Licorice 500 mg 90 chew
Price: $15.00
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DGL Licorice 500 mg 90 chew
Ingredients:
Licorice (DGL) 500 mg
Other Ingredients:
Fruit sugar
Beflora (Soy & Fos complex)
Steric acid
Suggested Use:
As a dietary Supplement chew one or two tablets just prior to each meal.
DGL Licorice 500 mg 90 chew
Ingredients:
Licorice (DGL) 500 mg
Other Ingredients:
Fruit sugar
Beflora (Soy & Fos complex)
Steric acid
Suggested Use:
As a dietary Supplement chew one or two tablets just prior to each meal.
Inactive Ingredients:
Beflora, Chicory Root, Fruit Sugar, Mung Bean Extract, Stearic acid
INGREDIENTS
Each chewable tablet contains:
Licorice (DGL) {Glycyrrhiza glabra root}
(standardized <2% {<10mg}glycyrrhizin) ……………….. 500mg
Recommended Usage: As a dietary supplement, chew 1 or 2 tablets just prior to each meal or
as recommended by your health care professional.
Rx Vitamin's Chewable Licorice is a standardized extract of deglycyrrhizinated licorice (DGL). It is
on the list of approved herbs by the German Commission E report. *
Licorice is a perennial herb native to Asia, the Mediterranean region and southern Europe.
Licorice is one of the most popular herbs worldwide, used for both medicinal and culinary
purposes. Historically it has been used as a sweetener, and medicinally for coughs, bronchial
ailments, adrenal insufficiencies and intestinal problems. The active components are glycyrrhizin,
glycyrrhetinic acid, flavonoids, isoflavones and triterpenoid saponins. One of the active
constituents, glycyrrhizin has a similar structure as the adrenal steroid hormones. This may
possibly cause sodium retention, potassium depletion, water retention and elevated blood
pressure. For this reason glycyrrhizin has been reduced, thereby eliminating the chances of side
effects. The deglycyrrhized form of licorice has many other active components and has shown to
be as effective (1-3).
With the widespread use of aspirin, one of the side effects (of aspirin) can be the development of ulcers (4).
And, one of the most common applications of licorice is to reduce the symptoms of ulcers (1, 5).
The deglycyrrhized form of licorice was found to reduce the number and size of ulcers that were
induced by aspirin consumption (1). A daily dose of 300 mg to 600 mg of deglycyrrhized licorice
was used to reduce gastrointestinal bleeding caused by aspirin (6). The method of action is not
totally understood but the different components seemed to have a healing affect on gastric
mucosa (7). The flavonoids found in licorice have been found to possess antimicrobial properties
(8). This antimicrobial activity leads to the theory that licorice may have an effect on H. pylori, the
gram-negative bacterium that causes some peptic ulcers.
Licorice has been known to exhibit anti-inflammatory as well as antiallergic activities (9). This
anti-inflammatory effect, along with the expectorant and demulcent action, may help reduce
coughs, bronchial spasms and congestion (3, 7).
Licorice has been known to exhibit anti-inflammatory as well as antiallergic activities (9). This
anti-inflammatory effect, along with the expectorant and demulcent action, may help reduce
coughs, bronchial spasms and congestion (3, 7).
Licorice has also been shown to reduce LDL cholesterol oxidation. The active components of
licorice inhibit the formation of lipid peroxides and protect LDL associated carotenoids (10, 11).
LDL oxidation is considered a major contributor to heart disease by increasing atherosclerosis
(hardening of the arteries). Bi-products of LDL oxidation are bioactive, and they secrete
cytokines, growth factors and cell surface adhesion molecules. In response to these oxidative biproducts,
smooth muscle cells proliferate in the wall of the artery, resulting in the narrowing of the
lumen and eventual blockage. Licorice may complement other nutritional supplements in reducing
LDL oxidation, atherosclerotic plaque formation and the risk factors for heart disease.
Licorice can be a valuable nutraceutical for gastrointestinal or bronchial disorders and may help
reduce symptoms of atherosclerotic plaque.
Drug Interactions: may interact with thiazide diuretics, digitalis or other heart medications.
Contraindications: do not use while pregnant or breast feeding, or if experiencing liver or kidney
disorders.
These statements have not been evaluated by the Food and Drug Administration. This product is
not intended to diagnose, treat, cure or prevent any disease
* In 1978 the German Federal Institute for Drug and Medical Devices established an expert
committee to evaluate the safety and efficacy of herbal remedies. This Commission E is made up
of physicians, pharmacists, toxicologists and lay people. They collected data on more than three
hundred herbal products from clinical trials, field studies, scientific literature and medical experts.
The data on the safety and efficacy was published in a report and is now used to recommend or
advise against using herbal remedies.
References:
1. Dehpour AR, Zolfaghari ME, Samadian T, Vahedi Y. The protective effect of liquorice
components and their derivatives against gastric ulcer induced by aspirin in rats. J Pharm
Pharmacol 1994;46(2):148-9.
2. Mowrey D. Next Generation Herbal Medicine. Lehi UT: Cormorant Books; 1988.
3. Roest RFM. Your Guide to Standardized Herbal Products. first ed. Prescott , AZ: One World
Press; 1995.
4. Physicians' Desk Reference. 53 ed. Montvale NJ: Medical Economics Data Production
Company; 1999.
5. Nadar TS, Pillai MM. Effect of ayurvedic medicines on beta-glucuronidase activity of Brunner's
glands during recovery from cysteamine induced duodenal ulcers in rats. Indian J Exp Biol
1989;27(11):959-62.
6. Rees WD, Rhodes J, Wright JE, Stamford LF, Bennett A. Effect of deglycyrrhizinated liquorice
on gastric mucosal damage by aspirin. Scand J Gastroenterol 1979;14(5):605-7.
7. Blumenthal M. The Complete German Commission E Monographs. first ed. Austin TX:
American Botanical Council; 1998.
8. Li W, Asada Y, Yoshikawa T. Antimicrobial flavonoids from Glycyrrhiza glabra hairy root
cultures [letter]. Planta Med 1998;64(8):746-7.
9. Kroes BH, Beukelman CJ, van den Berg AJ, Wolbink GJ, van Dijk H, Labadie RP. Inhibition of
human complement by beta-glycyrrhetinic acid. Immunology 1997;90(1):115-20.
10. Belinky PA, Aviram M, Fuhrman B, Rosenblat M, Vaya J. The antioxidative effects of the
isoflavan glabridin on endogenous constituents of LDL during its oxidation. Atherosclerosis
1998;137(1):49-61.
11. Belinky PA, Aviram M, Mahmood S, Vaya J. Structural aspects of the inhibitory effect of
glabridin on LDL oxidation. Free Radic Biol Med 1998;24(9):1419-29.
Manufacturer: RX Vitamins
SKU: EE-DGL3
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